Search results for "Metabotropic glutamate receptor 5"

showing 7 items of 7 documents

The Effects of N-Acetylcysteine on the Rat Mesocorticolimbic Pathway: Role of mGluR5 Receptors and Interaction with Ethanol

2021

N-acetylcysteine (NAC) is a prodrug that is marketed as a mucolytic agent and used for the treatment of acetaminophen overdose. Over the last few decades, evidence has been gathered that suggests the potential use of NAC as a new pharmacotherapy for alcohol use disorder (AUD), although its mechanism of action is already being debated. In this paper, we set out to assess both the potential involvement of the glutamate metabotropic receptors (mGluR) in the possible dual effect of NAC administered at two different doses and NAC’s effect on ethanol-induced activation. To this aim, 30 or 120 mg/kg of NAC was intraperitoneally administered to rats with the presence or absence of the negative allo…

0301 basic medicinePharmaceutical ScienceglutamatePharmacologyAcetylcysteine03 medical and health sciencesPharmacy and materia medica0302 clinical medicineNeurochemicalDrug Discoverymental disordersmedicinealcoholismMetabotropic glutamate receptor 5ChemistryCommunicationRGlutamate receptor<i>N</i>-acetylcysteineN-acetylcysteineRS1-441030104 developmental biologyMetabotropic receptorMTEPMechanism of actionMetabotropic glutamate receptorAlcoholismeMedicineMolecular Medicinemedicine.symptomTecnologia farmacèuticaMedicaments030217 neurology & neurosurgerymedicine.drugPharmaceuticals
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Endocannabinoid LTD in Accumbal D1 Neurons Mediates Reward-Seeking Behavior

2020

Summary The nucleus accumbens (NAc) plays a key role in drug-related behavior and natural reward learning. Synaptic plasticity in dopamine D1 and D2 receptor medium spiny neurons (MSNs) of the NAc and the endogenous cannabinoid (eCB) system have been implicated in reward seeking. However, the precise molecular and physiological basis of reward-seeking behavior remains unknown. We found that the specific deletion of metabotropic glutamate receptor 5 (mGluR5) in D1-expressing MSNs (D1miRmGluR5 mice) abolishes eCB-mediated long-term depression (LTD) and prevents the expression of drug (cocaine and ethanol), natural reward (saccharin), and brain-stimulation-seeking behavior. In vivo enhancement…

0301 basic medicineglutamate02 engineering and technologyMolecular neuroscienceBiologyNucleus accumbensMGLUR5 receptorsMedium spiny neuronArticleinduced reinstatementBehavioral Neuroscience03 medical and health sciencesDopamineDopamine receptor D2lipasemedicinelong-term depression[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]lcsh:ScienceLong-term depressionrelapseMultidisciplinaryMetabotropic glutamate receptor 5021001 nanoscience & nanotechnologyEndocannabinoid systemin-vivo exposure3. Good healthrats030104 developmental biologynervous systemethanol-seekingplasticitylcsh:Q[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Molecular Neuroscience0210 nano-technologyNeurosciencepsychological phenomena and processesNeurosciencemedicine.drugiScience
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Ontogenetic and Pharmacological Studies on Metabotropic Glutamate Receptors Coupled to Phospholipase D Activation

1997

The present study was aimed at characterizing the metabotropic receptor subtype which is involved in the activation of phospholipase D (PLD) by glutamate in rat hippocampal slices. We first observed that the ontogenetic profile of glutamate-induced hydrolysis of phosphoinositides and of phosphatidylcholine was strikingly similar. Both pathways were significantly activated by glutamate in tissue taken from 3-, 8- and 15-day old rats, but not in adult rats. PLD activation was strongest in slices taken from 8-day old rats. At this age, quisqualate had a higher potency for PLD activation (EC50: 0.6 microM) than 1S,3R-ACPD (EC50: 16 microM) and DHPG, a specific activator of group I mGluR, was a …

Agonistmedicine.medical_specialtymedicine.drug_classBiologyReceptors Metabotropic GlutamateHippocampusCellular and Molecular Neurosciencechemistry.chemical_compoundDCG-IVInternal medicinePhospholipase DmedicineAnimalsRats WistarPharmacologyDose-Response Relationship DrugMetabotropic glutamate receptor 5Glutamate receptorQuisqualic AcidRatsMetabotropic receptorEndocrinologychemistryMetabotropic glutamate receptorACPDMetabotropic glutamate receptor 1lipids (amino acids peptides and proteins)Neuropharmacology
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El modulador alostérico negativo de los mGluR5, MPEP, potencia la reinstauración de la preferencia condicionada inducida con priming de cocaína

2019

Cocaine addiction is a chronic disorder with high relapse rates; therefore, understanding the neuronal mechanisms underlying drug-seeking during relapse is a priority to develop targeted pharmacotherapy. The metabotropic glutamate receptor 5 (mGluR5) seems to be involved in the reinstatement induced by cocaine-associated cues. The main objective of the study was to evaluate the efficacy of MPEP, a negative allosteric modulator of mGluR5, in attenuating or potentiating the reinstatement induced by priming doses of cocaine in the CPP paradigm, ultimately to further knowledge regarding the role of the mGluR5 in relapse into cocaine abuse. OF1 mice (48 female and 48 male) were conditioned in th…

Allosteric modulatorMetabotropic glutamate receptor 5business.industryAddictionmedia_common.quotation_subjectMedicine (miscellaneous)Extinction (psychology)PharmacologyChronic disordersPsychiatry and Mental healthnervous systemmental disordersMedicinebusinessPriming (psychology)Cocaine abusemedia_commonAdicciones
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Expression of metabotropic glutamate receptors in murine thymocytes and thymic stromal cells

2000

RT-PCR combined with immunoblotting showed the expression of group-I (mGlu1 and 5) and group-II (mGlu2 and 3) metabotropic glutamate receptors in whole mouse thymus, isolated thymocytes and TC-1S thymic stromal cell line. Cytofluorimetric analysis showed that mGlu-5 receptors were absent in CD4(-)/CD8(-) but present in more mature CD4(+) CD8(+) and CD4(+)CD8(-) thymocytes. mGlu-1a receptors showed an opposite pattern of expression with respect to mGlu5, whereas mGlu2/3 receptor expression did not differ between double negative and double positive cells. mGlu receptors expressed in both thymic cell components were functional, as indicated by measurements of polyphosphoinositide hydrolysis or…

CD4-Positive T-LymphocytesMalemedicine.medical_specialtyStromal cellNeuroimmunomodulationReceptor expressionBlotting WesternImmunologyGene ExpressionThymus GlandCD8-Positive T-LymphocytesReceptors Metabotropic GlutamateCell LineMicePhosphatidylinositol PhosphatesInternal medicineCyclic AMPmedicineAnimalsImmunology and AllergyCycloleucineRNA MessengerReceptorReverse Transcriptase Polymerase Chain ReactionChemistryMetabotropic glutamate receptor 5HydrolysisMetabotropic glutamate receptor 6Flow CytometryCell biologyMice Inbred C57BLNeuroprotective AgentsEndocrinologyMetabotropic receptormetabotropic glutamate receptors; tc-1s cells; thymocytesNeurologyMetabotropic glutamate receptorMetabotropic glutamate receptor 1Neurology (clinical)Stromal CellsSignal TransductionJournal of Neuroimmunology
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Modulation of Neurological Deficits and Expression of Glutamate Receptors during Experimental Autoimmune Encephalomyelitis after Treatment with Selec…

2013

The aim of our investigation was to characterize the role of group I mGluRs and NMDA receptors in pathomechanisms of experimental autoimmune encephalomyelitis (EAE), the rodent model of MS. We tested the effects of LY 367385 (S-2-methyl-4-carboxyphenylglycine, a competitive antagonist of mGluR1), MPEP (2-methyl-6-(phenylethynyl)-pyridine, an antagonist of mGluR5), and the uncompetitive NMDA receptor antagonists amantadine and memantine on modulation of neurological deficits observed in rats with EAE. The neurological symptoms of EAE started at 10-11 days post-injection (d.p.i.) and peaked after 12-13 d.p.i. The protein levels of mGluRs and NMDA did not increase in early phases of EAE (4 d.p…

Encephalomyelitis Autoimmune ExperimentalMultiple SclerosisArticle SubjectHydrolasesEncephalomyelitislcsh:MedicineBiologyPharmacologyReceptors N-Methyl-D-AspartateGeneral Biochemistry Genetics and Molecular Biologymental disordersmedicineAmantadineAnimalsHumansRNA MessengerGeneral Immunology and MicrobiologyMetabotropic glutamate receptor 5Experimental autoimmune encephalomyelitislcsh:RGlutamate receptorMemantineGeneral Medicinemedicine.diseaseRatsDisease Models AnimalGene Expression RegulationReceptors Glutamatenervous systemCompetitive antagonistImmunologyNMDA receptorMetabotropic glutamate receptor 1FemaleExcitatory Amino Acid Antagonistsmedicine.drugResearch ArticleBioMed Research International
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Binge drinking upregulates accumbens mGluR5-Homer2-PI3K signaling: functional implications for alcoholism.

2009

The glutamate receptor-associated protein Homer2 regulates alcohol-induced neuroplasticity within the nucleus accumbens (NAC), but the precise intracellular signaling cascades involved are not known. This study examined the role for NAC metabotropic glutamate receptor (mGluR)-Homer2-phosphatidylinositol 3-kinase (PI3K) signaling in regulating excessive alcohol consumption within the context of the scheduled high alcohol consumption (SHAC) model of binge alcohol drinking. Repeated bouts of binge drinking ( approximately 1.5 g/kg per 30 min) elevated NAC Homer2a/b expression and increased PI3K activity in this region. Virus-mediated knockdown of NAC Homer2b expression attenuated alcohol intak…

Malemedicine.medical_specialtyReceptor Metabotropic Glutamate 5Binge drinkingContext (language use)Mice TransgenicNucleus accumbensReceptors Metabotropic GlutamateNucleus AccumbensArticleWortmanninchemistry.chemical_compoundMicePhosphatidylinositol 3-KinasesHomer Scaffolding ProteinsInternal medicinemental disordersmedicineAnimalsEthanolEthanolMetabotropic glutamate receptor 5business.industryGeneral NeuroscienceAntagonistUp-RegulationMice Inbred C57BLAlcoholismEndocrinologyPhenotypechemistryMetabotropic glutamate receptorbusinessCarrier ProteinsNeuroscienceSignal TransductionThe Journal of neuroscience : the official journal of the Society for Neuroscience
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